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Mare breasts

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If these signs are not present, odds are the mare does not have mastitis. However, she should be checked by your vet to make sure.

Mastitis can occur both in mares that are actively producing milk and those that are not. It is least common in a mare that is nursing a foal, unless there has been some type of injury to the teat.

Mastitis is often misdiagnosed in mares that have udder engorgement, which is a back-up of milk. Udder engorgement is common when a foal is weaned when it is still nursing frequently.

The udder is swollen and may be warm and sensitive to touch, but the fluid is obviously normal milk, with no clumps.

Udder engorgement may also be seen right before foaling and in the first day after foaling if the foal is not nursing well.

In fact, a full udder on a mare that has just foaled is an important clue to make sure the foal is up, alert and nursing, and that the mare is permitting him to nurse.

Mares with Cushing's disease, a benign tumor in the pituitary gland in the brain, often develop enlarged udders that may even leak milk.

This is due to overproduction of the hormone prolactin. Overweight and insulin-resistant mares that do not have a pituitary tumor sometimes also do this, and may have erratic estrus cycles or be difficult to get in foal.

It's not at all clear what is going on with these mares hormonally, but similar scenarios have been described in women.

Udder swelling without production of fluid can occur with viral diseases that produce edema along the belly and in the legs.

It may also occur with anything that blocks the free drainage of blood or lymphatic fluids from the udder. In a mare that is otherwise well, you should always check the area between the two sides of the udder and between the udder and the inner thigh for any signs of irritation if there is unexplained udder swelling.

Sweat, dirt and dead skin cells often accumulate in these areas and can inflame the skin. Be careful to not get kicked, since mares not used to having their udders examined can be touchy, and the skin irritation may cause the area to be extremely tender.

If you find this build-up of dirt and debris, treat the udder by cleaning gently with a sheath-cleaning product. Searching for the reason behind a mare?

A spanking-new foal may not be quite the picture you envisioned. However, it's important to recognize what's normal and what's not.

Learn in this article what to expect in the days ahead and at the time of birth. The foaling process is uneventful for most mares, requiring little, if any, intervention.

However, it's always important to be prepared, and to know what to watch for before, during and after birth. This article teaches owners the signs and behaviors of a cycling mare.

For optimal horse health and breeding success and owner safety, a horse owner needs to recognize when a mare is in heat.

Although your mare isn't cycling now, the care you give her today will ready her for conception in the months to come.

Learn why obesity is a real health risk, and what steps you can take to help your easy-keeping horse. Evaluate these aged mares' conformation and place them in your order of preference.

Then see how your choices compare with an expert horse judge's. Horse Breeds. Nearly 70 percent of women report breast pain at some point during their lives, but only around 15 percent need medical treatment.

The severity and location of breast pain can vary. Pain can occur in both breasts, one breast, or in the underarm. Severity can range from mild to severe and is typically described as tenderness, sharp burning, or tightening of the breast tissue.

Hormonal changes due to events such as menstruation, pregnancy, lactation, and menopause can also have an effect on the type of breast pain experienced.

Some women have a higher risk of developing painful breast conditions than others. At times, milk duct or gland changes may result in the formation of breast cysts.

Breast cysts can feel like a lump in the breast. They are fluid-filled sacs that can be soft or firm. They may or may not cause pain.

These cysts typically enlarge during the menstrual cycle and go away once menopause is reached. Most breast cysts consist of fluid rather than cells.

A cyst is different from a tumor , and it is not cancerous. Medications that are linked to an increase in breast pain include :.

Others that can have this effect include :. People with breast pain should speak to their healthcare provider to see if any of their medications are having an effect.

Undergoing breast surgery and the formation of scar tissue can lead to breast pain. The severity and type of pain vary between individuals and ranges from no pain to severe pain.

It can result from nerve damage or inflammation. Pain may affect the surface of the breast, or it may be deep. One study that followed up women who had undergone breast surgery over 6 months found that overall, mild pain tends to persist over time, moderate pain may increase, and severe pain will likely decrease.

Costochondritis, or costosternal syndrome, is an inflammation of the cartilage that connects the ribs and breastbone.

It can occur with arthritis. Arthritis in the neck or upper back can also lead to pain or numbness in the chest. It can also occur with an injury or physical strain.

Sometimes, there may be swelling. It is not related to the breast, but it causes a burning pain that can be confused with breast pain. Fibrocystic breast changes can cause one or both breasts to become lumpy, tender, and swollen.

This is due to a buildup of fluid-filled cysts and fibrous tissue. There may also be nipple discharge. This harmless condition is not uncommon in women aged between their 20s and 50s.

It is not linked to a higher risk of breast cancer. In stock now. To explore purchasing options for reusable face masks and non-surgical gowns, contact us at or info marenagroup.

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Stable Management. These alveoli join to form groups known as lobules. Each lobule has a lactiferous duct that drains into openings in the nipple.

The myoepithelial cells contract under the stimulation of oxytocin , excreting the milk secreted by alveolar units into the lobule lumen toward the nipple.

All the milk-secreting tissue leading to a single lactiferous duct is called a "simple mammary gland"; in a "complex mammary gland" all the simple mammary glands serve one nipple.

Humans normally have two complex mammary glands, one in each breast, and each complex mammary gland consists of 10—20 simple glands.

The presence of more than two nipples is known as polythelia and the presence of more than two complex mammary glands as polymastia.

Maintaining the correct polarized morphology of the lactiferous duct tree requires another essential component — mammary epithelial cells extracellular matrix ECM which, together with adipocytes , fibroblast , inflammatory cells, and others, constitute mammary stroma.

They not only help to support mammary basic structure, but also serve as a communicating bridge between mammary epithelia and their local and global environment throughout this organ's development.

A mammary gland is a specific type of apocrine gland specialized for manufacture of colostrum when giving birth.

Mammary glands can be identified as apocrine because they exhibit striking "decapitation" secretion. Many sources assert that mammary glands are modified sweat glands.

Mammary glands develop during different growth cycles. They exist in both sexes during embryonic stage, forming only a rudimentary duct tree at birth.

In this stage, mammary gland development depends on systemic and maternal hormones, [4] but is also under the local regulation of paracrine communication between neighboring epithelial and mesenchymal cells by parathyroid hormone-related protein PTHrP.

These mesenchymal cells can transform into a dense, mammary-specific mesenchyme, which later develop into connective tissue with fibrous threads, forming blood vessels and the lymph system.

These components of the extracellular matrix are strong determinants of duct morphogenesis. Estrogen and growth hormone GH are essential for the ductal component of mammary gland development, and act synergistically to mediate it.

During embryonic development, IGF-1 levels are low, and gradually increase from birth to puberty. Androgens such as testosterone inhibit estrogen-mediated mammary gland development e.

Mammary gland development is characterized by the unique process by which the epithelium invades the stroma. The development of the mammary gland occurs mainly after birth.

During puberty , tubule formation is coupled with branching morphogenesis which establishes the basic arboreal network of ducts emanating from the nipple.

Developmentally, mammary gland epithelium is constantly produced and maintained by rare epithelial cells, dubbed as mammary progenitors which are ultimately thought to be derived from tissue-resident stem cells.

Embryonic mammary gland development can be divided into a series of specific stages. Initially, the formation of the milk lines that run between the fore and hind limbs bilaterally on each side of the midline occurs around embryonic day The second stage occurs at E This will eventually give rise to the nipple.

Lastly, the third stage occurs at E The primitive stem cells are detected in embryo and their numbers increase steadily during development [32].

Postnatally , the mammary ducts elongate into the mammary fat pad. Then, starting around four weeks of age, mammary ductal growth increases significantly with the ducts invading towards the lymph node.

Terminal end buds, the highly proliferative structures found at the tips of the invading ducts, expand and increase greatly during this stage.

This developmental period is characterized by the emergence of the terminal end buds and lasts until an age of about 7—8 weeks.

By the pubertal stage, the mammary ducts have invaded to the end of the mammary fat pad. At this point, the terminal end buds become less proliferative and decrease in size.

Side branches form from the primary ducts and begin to fill the mammary fat pad. Ductal development decreases with the arrival of sexual maturity and undergoes estrous cycles proestrus, estrus, metestrus, and diestrus.

As a result of estrous cycling, the mammary gland undergoes dynamic changes where cells proliferate and then regress in an ordered fashion.

During pregnancy , the ductal systems undergo rapid proliferation and form alveolar structures within the branches to be used for milk production.

After delivery, lactation occurs within the mammary gland; lactation involves the secretion of milk by the luminal cells in the alveoli. Contraction of the myoepithelial cells surrounding the alveoli will cause the milk to be ejected through the ducts and into the nipple for the nursing infant.

Upon weaning of the infant, lactation stops and the mammary gland turns in on itself, a process called involution. This process involves the controlled collapse of mammary epithelial cells where cells begin apoptosis in a controlled manner, reverting the mammary gland back to a pubertal state.

During postmenopause , due to much lower levels of estrogen, and due to lower levels of GH and IGF-1, which decrease with age, mammary gland tissue atrophies and the mammary glands become smaller.

Lactiferous duct development occurs in females in response to circulating hormones. First development is frequently seen during pre- and postnatal stages, and later during puberty.

Estrogen promotes branching differentiation, [34] whereas in males testosterone inhibits it. A mature duct tree reaching the limit of the fat pad of the mammary gland comes into being by bifurcation of duct terminal end buds TEB , secondary branches sprouting from primary ducts [5] [35] and proper duct lumen formation.

These processes are tightly modulated by components of mammary epithelial ECM interacting with systemic hormones and local secreting factors. However, for each mechanism the epithelial cells' " niche " can be delicately unique with different membrane receptor profiles and basement membrane thickness from specific branching area to area, so as to regulate cell growth or differentiation sub-locally.

Elevated circulating level of growth hormone and estrogen get to multipotent cap cells on TEB tips through a thin, leaky layer of basement membrane.

These hormones promote specific gene expression. Hence cap cells can differentiate into myoepithelial and luminal duct epithelial cells, and the increased amount of activated MMPs can degrade surrounding ECM helping duct buds to reach further in the fat pads.

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